Introduction

This protrusion can subsequently trigger painful bursitis, a condition characterized by inflammation of the bursa, a fluid-filled sac located between the tendon and the bone.

It was initially described in 1927 by a Swedish orthopedic surgeon Patrick Haglund.

Patrick Haglund (1870-1937)

Bursitis results from constant irritation of the bursa, which can cause inflammation, redness and tenderness in the affected area. In addition to the characteristic bony bump, Haglund’s disease can cause persistent heel pain, especially when walking or using shoes that put pressure on the affected area.

Risk factors associated with Haglund’s disease often include anatomical problems, such as retroversion of the foot or abnormal gait, as well as frequent wearing of shoes that fit poorly or have a stiff back. Activities involving repetitive foot movements, such as running, can also aggravate the condition.

Treatment for Haglund’s disease generally aims to relieve pain and reduce inflammation. Non-invasive methods, such as the use of adapted shoes, heel lifts, icing and anti-inflammatory medications, are often recommended as first line treatment. In more severe cases or when symptoms persist, treatment options such as physiotherapy or surgical procedures may be considered to reduce irritation and correct the bone abnormality.

It is crucial to consult a healthcare professional for an accurate diagnosis and a treatment plan tailored to each individual, in order to minimize the impacts of Haglund’s disease on quality of life and mobility.

Causes of Haglund’s disease

Haglund’s disease, also known as “posterior heel protrusion” or “posterior calcaneal exostosis,” is a painful orthopedic condition that affects the heel region. This condition is characterized by a painful bony bump on the back of the heel, often associated with inflammation of the Achilles tendon and pain when walking or running.

One of the main causes of Haglund’s disease is the anatomy of the foot and ankle. People with a particular foot shape, such as pes cavus or a foot with an excessive tilt angle, may be more likely to develop this condition. This anatomy can cause increased pressure on the back of the heel, promoting the formation of the characteristic bony bump.

Additionally, inappropriate footwear can contribute to the development of Haglund’s disease. Shoes with a stiff heel or a design that puts excessive pressure on the back of the heel can worsen symptoms and encourage the bony bump to form. Shoes that rub or irritate the heel area can also cause inflammation of the Achilles tendon, making the condition worse.

Repetitive physical activities and repeated trauma to the heel may also play a role in the development of Haglund’s disease. Sports that involve flexion and extension movements of the foot, such as running or jumping, can put excessive pressure on the Achilles tendon and promote inflammation and bony bump formation.

Genetic factors may also contribute to predisposition to Haglund’s disease. Some people may have a genetic predisposition to developing bone abnormalities or orthopedic conditions, making them more likely to develop this condition.

Finally, factors such as age and body weight can also influence the development of Haglund’s disease. Older people, due to loss of flexibility and decreased tissue recovery capacity, may be more prone to orthopedic problems such as Haglund’s disease. Additionally, excess body weight can place additional pressure on the heel, exacerbating symptoms of the condition

  1. Foot Anatomy: Some individuals have foot anatomy that predisposes them to Haglund’s disease. This may include an outward tilt of the heel (retroversion) or other anatomical variations that increase pressure on the heel region.
  2. Abnormal Biomechanics: Biomechanical abnormalities, such as excessive pronation of the foot, can contribute to overload of the Achilles tendon and irritation of surrounding soft tissues.
  3. Inappropriate Footwear: Frequently wearing high-heeled shoes, especially those with a stiff back, can put constant pressure on the heel region, thereby promoting the development of Haglund’s disease.
  4. High Impact Activities: Participating in high impact activities, such as running, can increase stress on the Achilles tendon and contribute to tissue irritation.
  5. Heel Spurs: The presence of heel spurs, bony growths on the heel, may be associated with Haglund’s disease and contribute to the triad of symptoms.
  6. Genetic Factors: There may be a genetic component to the predisposition to Haglund’s disease, although research in this area is still ongoing.
  7. Overweight or Obesity: Excess weight can increase the load on the Achilles tendon and contribute to the development of Haglund’s disease.
  8. Predisposition to Inflammation: Some individuals have a predisposition to inflammation, which can worsen the inflammatory response in the heel area.
  9. Previous Trauma: Previous injuries, especially those involving the heel, can increase the risk of developing Haglund’s disease.
  10. Gender: Although Haglund’s disease can affect both sexes, some studies suggest it is more common in women.

Symptoms of Haglund’s disease

Haglund’s disease has a characteristic set of symptoms that may vary in intensity from person to person, but are generally similar in presentation. These symptoms are mainly localized in the heel region and can have a significant impact on the mobility and quality of life of those affected.

One of the most common symptoms of Haglund’s disease is a painful bump on the back of the heel. This bump, often described as a “Haglund’s bump,” is a bony exostosis that forms as a result of chronic irritation of the Achilles tendon and heel bone. The bump may be visible and palpable, and it is usually painful to the touch.

Another common manifestation of Haglund’s disease is pain in the heel, especially when walking, running, or any other activity that involves flexing or stretching the foot. This pain, often described as a shooting or burning pain, may be located at the back of the heel, around Haglund’s bump, and may radiate down the foot or along the Achilles tendon.

Symptoms of Haglund’s disease may also include inflammation of the Achilles tendon, called Achilles tendinitis. This inflammation can lead to increased stiffness and pain in the heel, especially in the morning upon waking or after a prolonged period of immobility. Movements that stretch or strain the Achilles tendon, such as climbing stairs or running uphill, can make symptoms worse.

Some patients with Haglund’s disease may also experience redness or swelling around the heel area, especially if there is severe inflammation of the Achilles tendon. This swelling may be associated with increased sensitivity to touch and may limit the range of motion of the foot and ankle.

Additionally, Haglund’s disease can cause deterioration in posture and gait, as individuals often attempt to relieve pain by changing the way they walk or stand. This alteration in posture can lead to additional muscle tension in other parts of the body, such as the knees, hips and lower back, thus worsening the symptoms of the condition.

  1. Pain at the back of the heel: Pain is one of the characteristic symptoms of Haglund’s disease. It is usually felt at the back of the heel, where the Achilles tendon attaches to the heel bone.
  2. Swelling: Inflammation of the soft tissues around the heel can lead to visible or palpable swelling in the affected area.
  3. Redness: The skin around the heel may become red due to inflammation, especially in cases of associated retrocalcaneal bursitis.
  4. Sensitivity to touch: The affected area may be sensitive to touch. Light pressure or wearing shoes can trigger discomfort.
  5. Noticeable bony bump: Haglund’s deformity, a posterosuperior calcaneal exostosis, may be palpable on the back of the heel.
  6. Increased pain when using shoes: Pain may become worse when wearing shoes, especially those that have a rigid back or that put pressure on the heel area.
  7. Possible limitation of foot mobility: Some individuals may experience a limitation of foot mobility, particularly when dorsiflexion of the foot.
  8. Symptoms Worsened by Activity: Pain and discomfort may intensify during activities such as walking, running, or climbing stairs.

Pathophysiology of Haglung disease

The pathophysiology of Haglund’s disease involves a complex combination of anatomical, mechanical, and inflammatory factors that contribute to the formation of the characteristic bony bump and associated symptoms. This orthopedic condition typically begins with chronic irritation of the Achilles tendon and heel bone, leading to a cascade of pathological responses.

At the heart of the pathophysiology of Haglund’s disease is the particular anatomy of the heel region. In some people, the shape of the heel and Achilles tendon may be such that there is increased pressure on the back of the heel, allowing chronic irritation and inflammation to develop. This excess pressure can be exacerbated by factors such as excessive foot pronation, high heel tilt angle, or being overweight.

Chronic irritation of the Achilles tendon and heel bone can lead to a local inflammatory response, characterized by the release of pro-inflammatory cytokines and chemical mediators. This inflammation contributes to the pain, swelling, and stiffness associated with Haglund’s disease. Additionally, it can stimulate excessive bone growth, leading to the formation of the bony bump at the back of the heel, called exostosis.

The formation of this bony bump can lead to a cycle of friction and continued irritation of the Achilles tendon, worsening inflammation and pain. Repeated friction between Haglund’s hump and the Achilles tendon can also cause additional injury and microtrauma to the tendon, compromising its structure and function.

Alongside these local mechanisms, Haglund’s disease may also involve alterations in the immune system and systemic inflammatory response. Studies have suggested a genetic predisposition to inflammation and excessive bone formation in some people, which may increase their susceptibility to developing Haglund’s disease.

Finally, certain external factors, such as inappropriate shoes or repetitive physical activities, can worsen Haglund’s disease symptoms by putting additional pressure on the heel area. Shoes with a stiff heel or a design that puts excessive pressure on the back of the heel can worsen Achilles tendon irritation and encourage bony bump formation.

The pathophysiology of Haglund’s disease can be understood by examining key steps in the process. Here is a step-by-step description of the pathophysiology of this condition:

  1. Predisposing Anatomy: Haglund’s disease is often associated with particular foot anatomy, such as an outward tilt of the heel (retroversion) or other anatomical variations. These anatomical features may create predisposing conditions.
  2. Mechanical Stress: Frequent use of inappropriate shoes, especially those with a rigid back such as high heels, generates excessive mechanical stress on the heel region.
  3. Achilles Tendon Irritation: Shoes cause constant friction between the heel and the shoe, irritating the soft tissues near the Achilles tendon. This can trigger inflammation and irritation.
  4. Retrocalcaneal Bursitis: Repeated irritation can lead to retrocalcaneal bursitis, characterized by inflammation of the bursa between the Achilles tendon and the heel bone.
  5. Achilles tendinopathy: Persistent inflammation and repeated mechanical stress can lead to Achilles tendinopathy, marked by degenerative changes in the tendon.
  6. Generalized Inflammatory Response: The pathophysiology of Haglund’s disease involves a generalized inflammatory response, affecting the surrounding soft tissues, including the Achilles tendon and bursa.
  7. Calcaneal Exostosis Formation: In response to continued traction of the Achilles tendon, a Haglund’s deformity forms in the form of a posterosuperior calcaneal exostosis, a bony bump at the junction of the Achilles tendon and the heel.
  8. Cycle of Damage and Repair: Persistence of mechanical stress and inflammation creates a cycle of damage and repair, with degenerative tissue changes that can be self-perpetuating.

Haglund’s triad

Haglund’s triad, is an orthopedic condition characterized by a combination of three main clinical features: Haglund’s hump, Achilles tendinitis, and retrocalcaneal bursitis. This triad of symptoms is often associated with chronic irritation of the Achilles tendon and heel bone, leading to a cascade of inflammatory responses and significant pain in the heel region.

The first component of Haglund’s triad is Haglund’s hump, a bony exostosis that forms at the back of the heel in response to chronic irritation of the Achilles tendon and heel bone. This bump, often described as a bony protrusion, can be visible and palpable, and is usually painful to the touch. It is caused by excessive bone growth in response to persistent inflammation and irritation.

The second component of Haglund’s triad is Achilles tendonitis, an inflammation of the Achilles tendon that manifests as pain, swelling, and stiffness in the heel. This inflammation is usually caused by repeated friction between the Achilles tendon and Haglund’s hump, thus worsening the symptoms of the disease.

The third component of Haglund’s triad is retrocalcaneal bursitis, an inflammation of the bursa located between the Achilles tendon and the heel bone. This bursitis develops in response to irritation and friction between the inflamed Achilles tendon and Haglund’s hump. It can lead to additional pain and swelling in the heel area, thus worsening the symptoms of the condition.

Haglund’s triad is often associated with activities that involve repetitive movements of the foot, such as running, jumping, or walking on hard surfaces. These activities exacerbate irritation of the Achilles tendon and promote the formation of Haglund’s hump, thus leading to worsening of the symptoms of the disease.

Diagnosis of Haglund’s triad usually relies on a thorough clinical examination, as well as imaging techniques such as x-rays, ultrasound or MRI to assess the extent of lesions and anatomical changes in the region of the heel.

Differential diagnoses of Haglund’s disease

  1. Plantar Fasciitis: Plantar fasciitis is an inflammation of the plantar fascia, the tissue that connects the heel to the toes. It can cause heel pain similar to that of Haglund’s disease.
  2. Isolated Heel Spur: A heel spur, a bony growth in the heel, may be independent of Haglund’s triad and cause similar symptoms.
  3. Posterior Compartment Syndrome: This condition involves increased pressure in the posterior compartment of the calf, which can cause heel pain and symptoms similar to Haglund’s disease.
  4. Heel Arthritis: Certain types of arthritis, such as rheumatoid arthritis, can affect the joints of the foot, leading to heel pain.
  5. Achilles Tendinopathy: Achilles tendinopathy can manifest with similar symptoms, although Haglund’s triad is specific to this condition.
  6. Isolated Retrocalcaneal Bursitis: Inflammation of the retrocalcaneal bursa, without the presence of Haglund’s triad, can also cause heel pain.
  7. Tarsal Tunnel Syndrome: This condition involves compression of the nerves in the ankle, which can cause heel pain and similar symptoms.
  8. Calcaneus Stress Fracture: A stress fracture of the heel can cause painful symptoms similar to those of Haglund’s disease.
  9. Posterior Pregnant Syndrome: This condition is characterized by pain at the back of the ankle, often involving the Achilles tendon.
  10. Upper Heel Bone Spur: A distinct bony growth in the heel can cause similar symptoms without the specific triad of Haglund’s disease.

Shoe that may cause Haglund’s disease

Choosing the wrong shoes can play a crucial role in triggering or worsening Haglund’s disease. Certain features of shoes can put excessive pressure on the back of the heel, leading to irritation of the Achilles tendon and the formation of Haglund’s hump. It is therefore essential to choose shoes that provide adequate support and reduce pressure on the heel region to prevent or alleviate the symptoms of this orthopedic condition.

High-heeled shoes are one of the main culprits that can contribute to Haglund’s disease. High heels change the alignment of the foot and ankle, tilting the heel forward and placing increased pressure on the back of the heel. This additional pressure can worsen the irritation of the Achilles tendon and encourage the formation of Haglund’s hump. Additionally, high-heeled shoes can overload the Achilles tendon, increasing the risk of tendinitis and retrocalcaneal bursitis.

Shoes with a stiff heel or a design that puts excessive pressure on the back of the heel can also cause problems for people predisposed to Haglund’s disease. These shoes limit the mobility of the foot and ankle, increasing friction between the Achilles tendon and the heel bone. This repeated friction can irritate the Achilles tendon and promote the formation of Haglund’s hump.

Shoes with a stiff rear construction or protruding seams can also cause additional friction and irritation in the heel area, worsening Haglund’s disease symptoms. These shoes can lead to inflammation of the Achilles tendon and retrocalcaneal bursitis, contributing to Haglund’s triad.

Finally, shoes that provide insufficient support or are not adapted to the anatomy of the foot can worsen the symptoms of Haglund’s disease. Shoes that do not provide proper arch support can cause the foot to overpronate, increasing pressure on the back of the heel. Likewise, shoes that are too tight or too loose can cause friction and irritation in the heel area, worsening the symptoms of the condition.

Exercise to do

Standing heel raise

Stand behind a chair. Place both hands on the backrest to balance yourself. Place your feet hip-width apart. Lift your heels off the floor, so your weight is on your toes. Do 10 repetitions. Frequency: 2 sets of 10 to 15 repetitions. Hold the weight in your hands, if it becomes too easy.

Stretching the Achilles tendon (calf):

Sit with your legs extended and your knees straight. Place a towel around your foot, just under the toes. Hold each end of the towel in each hand, with your hands above your knees. Pull the towel back so your foot stretches toward you. Hold this position for at least 15 to 30 seconds

Radiographic sign of Haglund’s disease

  1. Calcaneal Exostosis: One of the most obvious signs of Haglund’s disease on x-rays is the presence of a calcaneal exostosis, a bony bump on the back of the heel. This growth forms at the junction of the Achilles tendon and the heel bone.
  2. Haglund deformity: Calcaneal exostosis is often associated with a Haglund deformity, which is a posterosuperior bony outgrowth. This deformity is generally visible on lateral radiographs.
  3. Heel Spur: In some cases, heel spurs may be present, which manifests as additional bony growths.
  4. Abnormal Joint Space: X-rays may show signs of an abnormal joint space between the calcaneus and the posterior malleolus of the fibula.
  5. Widening of the Bony Cover: An x-ray may reveal a widening of the bony cover at the junction between the Achilles tendon and the heel bone.
  6. Assessment of Alignment: Radiographs also help assess the overall alignment of the foot and ankle, which can provide additional information about the pathophysiology of Haglund’s disease.
Bony prominence on the posterosuperior aspect of the calcaneal tuberosity with soft tissue swelling in the area. Courtesy of Dr Ayush Goel, Radiopaedia.org, rID: 62045
Bony enlargement on the posterosuperior surface of the calcaneus compatible with Haglund’s deformity. Marked edema of the surrounding soft tissues corresponding to insertional Achilles tendinosis and retrocalcaneal bursitis compatible with Haglund syndrome. Mild calcification of the Achilles insertion is noted. Similar but less severe insertional tendinosis and calcification are seen on the left without Haglund’s disease. Courtesy of Dr Andrew Dixon, Radiopaedia.org, rID: 28562

Conclusion

In conclusion, Haglund’s disease is a complex and debilitating orthopedic condition characterized by the presence of a painful bony bump on the back of the heel, often accompanied by Achilles tendonitis and retrocalcaneal bursitis. This condition can lead to persistent pain, inflammation and impaired function of the foot, impacting the quality of life of those affected.

The pathophysiology of Haglund’s disease involves a complex interplay between anatomical, mechanical, inflammatory, and genetic factors, contributing to the formation of Haglund’s hump and the associated triad of symptoms. Inappropriate shoes, such as those with high heels, stiff heels, or those with insufficient support, can make symptoms worse by increasing pressure on the back of the heel and causing irritation of the Achilles tendon.

Early recognition of Haglund’s disease symptoms and appropriate management are key to relieving pain, reducing inflammation, and preventing long-term complications. Treatment options may include conservative measures such as rest, ice, stretching, wearing appropriate shoes, and physical therapy, as well as more invasive options like corticosteroid injections or surgery in severe cases.

Additionally, a preventative approach focused on choosing appropriate footwear and modifying activities that exacerbate symptoms can help reduce the risk of developing or worsening Haglund’s disease.

Ultimately, a thorough understanding of the pathophysiology, symptoms and risk factors associated with Haglund’s disease is essential to provide patients with optimal care and improve their quality of life. By raising public awareness of the implications of inappropriate footwear and encouraging proper footwear practices, it is possible to reduce the burden of this debilitating orthopedic condition.

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